For example, low catalytic efficiencies may be poorly estimated in the range appropriate for high catalytic efficiencies and a higher/broader range may be required to distinguish catalyzed from intrinsic exchange.
In exposure response models, individual exposure (e.g., AUC from Dose/CL) may be poorly estimated when shrinkage in CL is high, thereby lowering power to detect an exposure response relationship.
A possible explanation is that under the IM and the traditional polygenic model, the Bayes B method will use information from a number of selected SNPs, and although the effects may be poorly estimated and a random set of markers is used, the resulting prediction is similar to gBLUP.
Therefore the parameters associated with potential cross-level interactions may be relatively poorly estimated and we may not have detected a significant effect where one existed.
The fact that θ1 is more poorly estimated than θ0 may seem counterintuitive as one might expect it to be easier to estimate parameters for recent ancestors (such as θ1) than for ancient ancestors (such as θ0).
Hence, traditional QTL mapping approaches that ignore the phenotype network result in poorly estimated genetic architecture of phenotypes, which may hamper correct inferences regarding causal relationships among phenotypes.
Rainfall was the most poorly estimated variable, and in some cases, the error may become very important in models, particularly in dry areas where water supply is the dominant factor influencing growth.
This may mean that the relative location errors, especially those for focal depth, are poorly estimated.
Therefore, the wrong ambiguity is poorly estimated, resulting in a large gap in delay.
Branch dry weight (MAPE = 80%) and foliage dry weight (MAPE = 83%) were poorly estimated.
