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This result indicates that the N-terminal deletion may increase enzyme activity.
These domains may increase enzyme efficiency by enhancing the localization of enzymes on the surface of crystalline cellulose [ 50] or disturb the structure of crystalline cellulose [ 51].
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Low constitutive TPI expression in the host plant may increase proteolytic enzyme activity in the guts of neonates, digestion efficiency and the growth rates [ 44] (Fig. 4).
Acetylcholinesterase (AChE) enzyme is involved in the breakdown of acetylcholine in the brain and inhibition of this enzyme may increase the efficacy of treatment and broaden the indications [ 6].
Overexpression of single gene encoding a key enzyme may increase flux through the pathway, but its effect may be limited by other rate-committee step to some degree.
Dual inhibition of these enzymes may increase the efficacy of treatment and broaden the indications.
Indeed, defects in the activity of mitochondrial antioxidant enzymes may increase oxidative stress.
A recent large RCT demonstrated that liberal oxygen administration during the acute phase of myocardial infarction may increase the level of cardiac enzyme and the infarct size [2].
The presence of insufficient antioxidant enzyme system may increase the susceptibility to oxidative stress-associated diseases among south Indian population.
However, it remains unclear if genetic differences in drug metabolizing enzyme expression may increase the risk of SSZ toxicity.
The interaction between the surfactant and the enzyme is known to be hydrophobic (Maldonado-Valderrama and Patino 2010) that may increase the interfacial surface of the enzyme.
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