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Prebiotics may increase colonization by lactobacilli and can prevent mucosal inflammation.
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For example, some pathogenic Candida species aggregate with Fusobacterium sp. [ 32], Actinomyces sp. [ 33], which may increase the colonization of mucosal epithelial cells.
Our data indicate that the elimination of nasopharyngeal colonization with bacteria such as S. pneumoniae and H. influenzae may increase risk for colonization with S. aureus.
We suggest that breeding for elevated concentrations of BXs in maize may have unintentionally increased colonization by Fusarium species.
Therefore, we can not exclude that rates of multiple colonization may increase in the future with the emergence of these serotypes.
Factors that may increase the risk of colonization with S. aureus are of special concern given the spread of methicillin-resistant S. aureus (9 ).
Pneumocystis colonization may increase the risk for progression to PCP, carriers of the organism may transmit infection to others, and latent infection may lead to inflammation that is detrimental to the lung.
Just as vaccination programs benefit those who are not vaccinated because of the phenomenon of herd immunity, antibiotic usage by some persons may increase the risk of colonization or infection with resistant organisms in people who have not received antibiotics.
Treatment of respiratory Candida spp. colonization in non-neutropenic critically ill patients by inhaled ABDC may not affect respiratory colonization but may increase duration of mechanical ventilation, because of direct toxicity of the drug on the lung.
In addition, protein malnutrition (i.e. low serum albumin) may increase the patients' susceptibility to MRSA colonization [ 19].
While catheter tip colonization and CRI may increase with CVC duration controversy still exists regarding scheduled changes.
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