The explanation for these observations are uncertain but contributing factors may also include underlying differences in transmission dynamics since patients with febrile neutropaenia and central line infections typically have more previous healthcare exposure than patients with bacteraemia following an outpatient prostate biopsy.
The broad extent of tissue, cell type, developmental and pathological expression annotation suggests that these sequences may include sequences underlying tissue and organ development as well as contributing to specific pathological conditions.
These differences may reflect a range of factors, including underlying prevalence, rates of diagnosis and care seeking, and proportions with drug versus non-drug treatment.
This may include those with underlying thromboembolic disease or during pregnancy [75, 77].
In humans, these may include genes that underlie generosity, moral constraints, even religious behavior.
However the mortality data and the OR to calculate the sample sizes are not for an immunocompromised population and may include confounding effects of underlying diseases and severity.
Potential mechanisms for this rebound may include a homeostatic pressure underlying Slow Wave generation [40] or a reversal of the effect of stimulation from inhibitory to excitatory due to the sustained nature of stimulation [41], [42].
Underlying reasons may include an oceanic buffering effect that attenuates increases in air temperature, and the complex topography that generates micro-refugia.
This may include increasing knowledge about the underlying philosophy and history of MMR, examples of MMR research designs and principles, and the pros and cons of this approach above a purely qualitative or quantitative inquiry.
The underlying mechanisms may include not only the intrinsic electrical and biochemical properties of the axon itself [5], [49], but also active communication between axons and their local environment and neighboring glia [50] [52].
