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Additional photocoagulation may improve retinal ischemia; here it may have led to the satisfactory surgical outcomes.
These findings highlight the importance of close blood pressure monitoring by all professionals involved in the management of adolescents with diabetes, regardless of urinary albumin excretion and glycaemic control, and indicate that lowering blood pressure, even in patients without hypertension or microalbuminuria, may improve retinal outcomes in diabetes.
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In translational research, if this decoupling phenomenon is also found in the retina in vivo, it could indicate that targeting diabetic retinal neuropathy by enhancing cellular reducing ability or with antioxidant agents may not necessarily improve retinal neuronal survival in diabetic retinal neuropathy.
The AF ablation may improve a retinal function, which was registered on OPs WI.
The AF ablation may improve the retinal function as indicated by an increase in the OPs WI.
Although the mechanism of improved vision cannot be proved, it is probable the reduction in IOP from the very high preoperative levels may have improved retinal ganglion cells function through restoration of axoplasmic flow and improved microcirculation to the optic nerve.
We hypothesize that in the diabetic milieu with neuroretinal hypoxia (5), early bifurcation may be a compensatory mechanism to increase vessel density and improve retinal perfusion (perhaps the earliest form of neovascularization).
Intravitreal injection of bevacizumab may improve visual acuity, decrease retinal thickness and reduce angiographic leakage in AMD [ 19].
Our finding of a non significant two-fold increase of vasodilation suggest that, if any, vasodilatory capacity of flicker light (a parameter of early vascular remodeling of the retinal arterioles) may improve after DPP-4 inhibition with saxagliptin.
Triamcinolone acetonide may improve macular edema by decreasing retinal capillary permeability via changes of tight junctions [ 20], or it could inhibit the signaling cascade involving VEGF and its receptor that increases microvascular permeability [ 21, 22].
Considering the contradictory phenomena of low log P value and the high penetration of F Tau, we may reasonably conclude that the transport of F Tau is mediated via a specific transporter, and that this transporter may improve the penetrability of fluorescein to retinal cells.
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