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While these inputs are normally overshadowed by V1 connections, acknowledging their contribution may improve neuronal models.
GEB contains various biologically active components [ 25] that may improve neuronal cell viability through inhibition of apoptosis [ 24, 26].
Based on these evidences, strategies to either increase the endogenous upregulation or supplement it with exogenous IGF-1 may improve neuronal survival after TBI.
Strategies to either increase the endogenous upregulation of IGF-1 after TBI or supplement it with exogenous IGF-1 may improve neuronal survival after TBI [ 23].
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Thus, GDAsBMP not only support survival of different neuronal populations but also may improve neuron functionality.
In addition, inhibition of neuronal excitability by SKF83959 may also improve neuronal survival and contribute to its neuroprotective effect.
Cellot, G. et al. Carbon nanotubes might improve neuronal performance by favouring electrical shortcuts.
Consequently, defects in neuronal metabolism may exist regardless of the provision of alternative energy substrates (e.g. through high calorie feeding [ 14, 50, 53, 62, 144] or ketogenic diet [ 145]) to sustain or improve neuronal energy supply.
Striatal dopamine transporter (DAT) imaging is a sensitive biomarker of substantia nigra pars compacta (SNc) neuronal density that may improve the accuracy of in vivo diagnosis in patients with CBS, given that SNc cell loss is a pathological finding considered to be mandatory for the definite diagnosis of CBD [3], [16], [17].
Our findings suggest that aged garlic extracts with antioxidant activities may improve cognitive impairment against Aβ-induced neuronal deficit, and possess a wide range of beneficial activities for neurodegenerative disorders, notably Alzheimer's disease (AD).
Recently, alcohol dehydrogenase 5 (ADH5) is found to be a novel suppressor of neuronal differentiation and maturation, suggesting that ADH5 may improve adult neurogenesis in a physiological or pathological setting (Wu et al., 2014).
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