Behind the assumption that increased stress may explain declining sleep quality is the large body of evidence indicating the negative effects of stress on sleep [18], [24], [32], [34], [36], [38], [42], [43].
There are several potential pathways, both biological and behavioral, that may explain how sleep problems duration may be related to overweight/obesity.
Another explanation could be the effect of antipsychotic medication, which may explain increased sleep time, irrespective of group, and decreased daytime activity due to sedation or effects on motivation.
This sleep disruption may explain the overwhelming fatigue, report of unrefreshing sleep, and pain in this subgroup of patients.
This sleep disruption may explain the overwhelming fatigue, report of unrefreshing sleep, and pain of patients in this subgroup.
New research suggests that a genetic mutation may explain why some people sleep less.
The role of the autonomic nervous system in the stimulation of the adaptive immune response may explain why mood and sleep have an influence on antibody production.
Several potential mechanisms may explain the associations between sleep disorders, mental health conditions, and diabetes risk.
It is also possible that a decline in cerebral and systemic glucose use (34) and counter-regulatory hormone release during sleep (35) may explain the association between the elevation of the HbA1c levels and extended sleep.
There are several potential factors that may explain the increased persistency of sleep problems in the chronic illness group.
