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Such studies appear especially needed since A. artemisiifolia was long reported as self-compatible [15],[68] and Friedmann & Barrett [19], who showed the self-incompatibility in Canadian populations, acknowledged the possibility that some other populations may exhibit partial self-compatibility.
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Therefore, functionally TFs and co-factors may exhibit a partial redundancy even when their exact biochemical activities and ranges of interaction partners do not completely overlap 33– 37. Further, this model suggests that transcriptional activation does not have to originate from a single TFBS or even a single regulatory module containing multiple TFBSs.
The ecotypes also exhibit partial, but incomplete, progress towards ecological speciation [33], [72], [75] [80].
Whereas I4-FFL-A achieves optimal adaptive response, it could be argued that the suboptimal circuits exhibit partial adaptation.
Indeed, EGFL7 loss-of-function mouse models exhibit partial embryonic lethality and serious vascular abnormalities [ 7].
Half of the intermolecular hydrogen bonds exhibit partial proton transfer within the FE-I phase.
The 3 relaxing tasks (Relax0, Relax1, and Relax2) exhibit partial internal confusion.
This suggests that some mutants analyzed here (and in previous mutant library screens) may exhibit phenotypes because of partial deletions of overlapping verified genes, not because of the ORF whose coding region was deleted.
When the harmonicity is not ideal, the overlapping continues since the partials of the notes may exhibit appreciable bandwidth.
Therefore, these mice may exhibit an unexpected behavioral phenotype resulting from the partial reductions of DAT and NR1 expression levels.
Intriguing is the finding that, although primary ductal and lobular breast cancers can show partial or complete loss of E-cadherin expression, their derivative metastases may exhibit strong E-cadherin expression [ 23, 24].
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