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Enhancing circulating EPC may enhance vascular protection and reduce the progression of CVD.
Therefore, VEGF in SRMA may enhance vascular wall destruction in the acute phase of the disease before immune complexes appear [ 33].
However, once the bud start to grow, buds synthesize auxin, as observed in other plants [ 85], and its export may enhance vascular connections and nutrient flow to further stimulate the growing bud.
Moreover, CD73 may enhance vascular permeability by induction of adhesion proteins on endothelial cells [ 39] and the extracellular fluid of solid carcinomas was shown to contain biologically active concentrations of adenosine [ 40].
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Contrarily, leptin may also enhance vascular constriction.
It seems that CSVD could stimulate amyloid pathology, while AD-associated cerebral amyloid pathology may enhance auxiliary vascular damage.
Improved levels of HbA1c by pharmacological therapy may lead to an increase of BM-CPCs frequency and functional activity and thereby may enhance the vascular regeneration in IHD patients with DM.
This study suggests that heparin binding growth factors may be able to enhance vascular healing and remodeling of heparin bound biological grafts.
In theory, alcohol may affect platelet function, coagulation physiology, and enhance vascular fragility [ 23].
However, since arrestin proteins can enhance vascular smooth muscle migration and proliferation, their increased expression may potentially exacerbate vasoconstrictor-mediated vascular remodeling and hypertension.
These and other chemical species may enhance pulmonary and vascular effects.
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