Mechanisms that enable prostate cancer cells to evade apoptosis may contribute to therapeutic resistance.
Further functionalization by addition of tumor-specific ligands may enable GNPs to further target prostate cancer with increased specificity.
On these, we hypothesized that GPR110 expression may enable us to differentiate between benign prostate hyperplasia (BPH) and potential incipient malignancy.
Furthermore, a biostatistical model was developed using a limited number of molecular markers that may enable more accurate prediction of risk of prostate cancer progression.
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Each may enable or constrain the partnership.
This may enable effective disease management.
Novel injury biomarkers may enable earlier diagnosis.
A miR-based test may possibly enable clinicians to distinguish more accurately indolent or non-metastatic prostate cancer from aggressive disease with metastatic potential.
A number of modalities exist that enable localized prostate ablation.
