In theory, frequent or prolonged exposure to nicotine absorbed from passive smoke may predispose nicotine-naive adolescents to develop nicotine dependence once they initiate smoking.
Evidence from affected relative or allele sharing methods of analysis demonstrates a number of plausible "candidate genes" for nicotine dependency, which may affect individual's vulnerability to develop nicotine dependency.
(Only 0.4percentt of seniors reported use in the last year.) Still, researchers are concerned that vaping can lead to more drug use and that some teens may be developing nicotine dependencies without even realizing.
"We are concerned that e-cigarettes may encourage nonsmokers, particularly children, to start smoking and develop nicotine addiction".
Furthermore, individuals susceptible to nicotine poisoning could avoid tasks with higher exposure or leave tobacco work entirely (healthy worker effect), and some workers may develop tolerance to nicotine [Arcury et al., 2001a].
Symptoms of nicotine dependence may develop soon after initiation even at low levels of smoking (Zhan et al, 2012); thus, slower nicotine clearance at adolescence may be associated with greater addiction levels, perhaps mediated by a greater number of cigarettes smoked (Rubinstein et al, 2013).
Parental smoking may shape cognitive responses to tobacco use (e.g., through outcome expectancies) as well as actual smoking behaviour (e.g., through social modelling or social norms), which may enhance adolescents' risk of developing nicotine dependence.
"Alternate identities may develop instead".
With time, though, differences may develop.
One day a planned interdependence may develop.
