K. and E. S., unpublished observations), it is likely that transgenic mice with low level miR-130a expression may also not develop cardiac malformations due to only modest reductions in FOG-2 protein levels.
Athletes may develop structural cardiac changes with a steeper slope of the left cardiac pressure stroke volume relationship indicating a greater reduction in stroke volume for a given decrease in pressure during an orthostatic challenge (Fu et al. 2004).
Patients with heart disease may decompensate due to the physiologic changes that occur during pregnancy and may develop a cardiac event with obstetric events.
Introduction: During weaning from invasive mechanical ventilation (IMV), pulmonary oedema may develop due to cardiac failure, and it is a cause of failure in weaning from IMV. Cardiogenic pulmonary oedema produces hemoconcentration and haematocrit (Ht) is a good marker of hemoconcentration.
Athletes with EIAH are in similar way "persons at risk" and may develop a pathological cardiac chamber enlargement and atrial fibrillation, but have less "cardiovascular risk" because of the healthier life style and the positive impact of sport in the development of arteriosclerotic complications.
Decades after surviving the potentially deadly acute phase of Chagas disease, a human infected with T. cruzi may develop fatal damage to cardiac and smooth muscle tissue in approximately 30% of cases.
Following cure of acromegaly with surgery or radiation therapy, GHD may develop; however, its effects on cardiac morphology and function in this population are not established.
The failing heart may develop a "hypothyroid" status contributing to diminished cardiac contractile function.
After cardiac surgery, pulmonary dysfunction may develop as shown by a significant reduction of functional residual capacity (FRC) after extubation [ 1].
Late cardiac complications in cancer survivors may develop from subclinical myocardial damage.
