More mature embryos may have a rudimentary procambial trace, although may not demonstrate normal patterning (Fig. 3G).
Although we lack a complete explanation, this may demonstrate the importance of normal tissue damage in determining long-term survival.
Initial CT studies are often normal, or may demonstrate subtle temporal lobe hypodensity.
Imaging may be normal or may demonstrate cerebellar dysplasia, hemispheric hypoplasia or atrophy, neuronal dysplasia, periventricular hyperintensity or intracerebral haemorrhage (Baxter, 2001).
It is understandable that transitional molecular changes represented by gene sets may demonstrate mechanistic trend of development from normal tissue to cancer tissue, however, whether such changes can be prognostic may be another question.
Previous studies have suggested that 33% of adjacent normal prostate tissue may demonstrate some raised expression of PRDX-3, consistent with a field effect seen in other solid tumours (Basu et al, 2011).
With lysate from a better batch of protein expression there is a possibility that all of the sequences may demonstrate higher binding reactivity to tumor than normal sera.
Therefore, these lesions have fewer macrophages, including Kupffer cells, than normal liver tissue, and these lesions may demonstrate a defect in the Kupffer phase.
Pineal parenchymal tumours often produce an "explosion" of normal pineal calcifications towards the periphery and may demonstrate areas of cystic changes.
In selected patients who previously have undergone YAG laser ablation of the endometrium and who have demonstrated normal intrauterine architecture, pregnancy as a planned event may be a reasonable and safe option.
