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Allopolyploidy plays an important role in plant evolution that arises with the merging of two or more genomes into single nucleus which may contribute either equally or disproportionately.

Therefore, depending on the cell type and tumour model, expression of various integrin subunits may contribute either positively or negatively to the transformed phenotype.

Although these cations do not play a direct role in the cleavage mechanism, they may contribute either through electrostatic stabilization or by modulating the p Ka of functional residues.

Together, the consistent expression of ECM gene family members in human CTCs suggests that their upregulation may contribute either to the generation of CTCs from primary tumors or to the survival of cancer cells deprived of microenvironmental signals as they circulate in the bloodstream.

Thus decreased JNK activation may contribute, either directly or indirectly, to earlier CXCL10 gene transcription in the asthmatic cells.

However, other forms of cellular plasticity may contribute, either within the cerebellar cortex (Hansel et al., 2001; Carey, 2011) or in the deep cerebellar nucleus (Miles and Lisberger, 1981; Zhang and Linden, 2006; Zheng and Raman, 2010).

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Reduced CPI activity within the CSF of ALS or other neurologic disease patients may contribute to either toxic or protective pathways related to disease pathogenesis.

5– 7 The abnormal expression of miRNAs may contribute to either tumor progression (oncomirs) or tumor regression (tumor suppressor) through miRNA mRNA interactions (MMIs).

Apoptosis is a tightly controlled suicide program that serves to remove defective, damaged, or otherwise superfluous cells, whereas autophagy may contribute to either cell survival or cell degradation.

Although both food activities and identity maintenance contribute to active ageing and improved quality of life, little is known about how food activities may contribute to either maintenance or change in identities in later life.

Several studies indicate that an inflammatory process in the SN, characterized by the presence of activated microglia and the secretion of pro-inflammatory and neurotoxic factors, may contribute to either initiation and/or aggravation of neuronal degeneration.

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