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They suspected that spinal anesthesia may cause neuronal ischemia [17] or infarction owing to changes in endoneurial small vessels, blocking of sodium channels in demyelinated areas or interaction of regional anesthesia with peripheral myelin or nerve trauma, which may result in deterioration of symptoms.
This reduction may cause neuronal vulnerability.
An expanded PolyQ tract may cause neuronal dysfunction by interrupting the transcriptional enhancer function of α1ACT.
Decreased oxygen supply to brain may cause neuronal damage in COPD.
Activated astrocytes, especially through increased oxidative stress by alcohol, may cause neuronal damage.
Here, we identified TGF- β1-induced antiapoptotic factor (TIAF1) protein aggregation, which may cause neuronal death.
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Mechanistically, it has been suggested that 3-OMD may interfere with the utilization of L-DOPA in the rat brain, and it may also cause neuronal damage via oxidative stress.
3 6 Moreover, systematic inflammation 1 may also cause neuronal damages in the brain of patients with COPD.
However, reactive gliosis may cause secondary neuronal damage as major neuron-supportive functions of glial cells get lost.
Therefore, acetone cyanohydrin may cause selective neuronal degeneration in the rat, but the affected areas are not those expected in an animal model of konzo.
An absence of neurotrophins suppresses Trk receptor expression and may cause cognitive neuronal defects.
More suggestions(15)
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