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Since the found associations are independent of symptom level they may be trait variables.
A meta-analysis of cytokine alterations in schizophrenia showed that molecules such as IL-1β, IL-6 and TGF-β may be state markers for acute exacerbations, whereas others, including IL-12, IFN-γ, TNF-α and soluble IL-2 receptor, may be trait markers [ 40].
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On the other hand, our results seem to indicate that defective ability to apply others' mental states may be trait-dependent.
Therefore, we preliminarily conclude that deficits in the ToM ability to attribute others' mental states may be state-dependent, whereas deficits of the ToM ability to apply knowledge of others' mental states may be trait-dependent.
This, and the fact that GBLUP0 was slightly more accurate than GBLUP20 for some traits, suggests that the optimal weight (w) for pedigree-based relationships in this alternative implementation of GBLUP may be trait-specific, as pointed out by [ 43].
In addition, for all traits, expected accuracies were based on the assumption that markers explain 80% of the genetic variance, and the adequacy of this assumption may be trait-specific.
The thinning may be a trait or a marker of vulnerability to depression, the researchers suggested.
Therefore, the H2S reducing activity of SJP-SNU may be valuable trait as a probiotic.
Results suggest that hippocampal volume reduction may be a trait marker for identifying individuals possessing a genetic predisposition for schizophrenia.
The ability to communicate through spoken language may be the trait that best sets humans apart from other animals.
Hence, endurance capacity may be the trait that underlies both physical activity levels and leanness.
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