In addition, immunofluorescence staining suggested that HIF-1α immunoreactivity was colocalized with NeuN immunoreactivity, indicating that the effect of EA pretreatment on HIF-1α expression may be neuron-specific.
Additionally, the source of the Aβ peptides that produce deposits may be neurons that project axons into the hippocampus as well as local neurons.
These data show that P1 neurons receive sensory cues signaling females or males and drive courtship decisions, suggesting that they may be command neurons for courtship behaviors.
Because of this lack of specificity for the peptide presented, any conformationally correct sMHCI is a potential ligand, which may be why neurons are sensitive to such low levels of sMHCI.
Given their location and morphology, the lateral a2-a4 cells may be serotonergic neurons.
On the other hand, the GnRH antibodies may be secondary to neuron degeneration and subsequent exposure of neuron material to inflammatory cells.
Similar to previous optetrode studies (e.g., Cohen et al., 2012), some actual AgRP neurons may be classified as ARCother neurons in cases of insufficient photostimulation.
We believe that the transcriptional expression of GIRK may be expressed in neuron or interstitial cells.
This pattern suggests that some of the labeled neurons from the dorsal and dorso-medial groups may be multiglomerular projection neurons that receive input from several glomeruli [54].
If these neurons acquire similar selectivity through their connections, a salt-and-pepper organization may be generated, because neurons derived from different progenitors are intermingled in rodents.
Alternatively, these neurons may be strongly connected to motor neurons that control effectors on the same side of space.
