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After a viral illness, there may be inflammation around the airways that causes cough and wheezes.
A growing number of medical researchers are coming around to the idea that the common denominator of many, if not most, of the chronic diseases from which we suffer today may be inflammation — a heightened and persistent immune response by the body to a real or perceived threat.
Another explanation may be inflammation of the optic nerves due to the infected adenoma [ 6].
However, the mechanism by which effusion is generated is unclear; the cause may be inflammation initiated by disseminated Penicillium marneffei infection, or may directly involve the thoracic cavity.
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Possible explanations for this process may be inflammation-driven endosteal resorption, which was also found in human juvenile idiopathic arthritis [ 35], experimentally induced inflammatory knee arthritis or adjuvant-induced arthritis [ 36- 38].
During this initial phase there may be transient inflammation of the heart muscle, but usually with little or no impairment of the circulation.
Follow-up culture and universal PCR were negative suggesting this may be recurrent inflammation alone.
Another explanation for the relationship between RDW and prognosis may be chronic inflammation.
Specifically, caffeine may be blocking inflammation rather than fibrosis directly, since adenosinergic signaling in inflammatory cells is well-established.
Inflammation in emphysema may be different from inflammation in airway disease [ 50], explaining the variable correlations between these COPD phenotypes and other diseases associated with systemic inflammation.
Iron studies may be affected by inflammation, and we have limited ability to assess such inflammation.
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