Sentence examples for may allow binding from inspiring English sources

This may allow binding and cross linking both intramolecularly to two of the three units of the viral spikes, intermolecularly to different spikes on a virus or by agglutination of the viruses.

Receptors such as HSPG and the 32 k laminin receptor that are abundant at this site may allow binding of AAV serotypes 2, 8, and 9, whereas AAV serotypes such as 1 and 5 that bind sialic acid, which is not found at the ILM, show no expression in the rat retina following intravitreal administration [15], [25], [40].

This reflects the in vivo situation and indeed may allow binding of high-affinity as well as low-affinity T cells [ 52].

In addition, optimal ligand concentrations leading to formation of immune complexes may allow binding to FcγRIIIb and subsequent activation of cells.

It is possible that a loss of H3K9 methylation may have been induced by 5-aza-dC in the current study, which may allow binding of transcriptional proteins, which in turn enhance transcriptional reactivation.

The apposition of ICAM-5 labeled elements with dendritic and axonal profiles may allow binding to the α5β1 integrin in both pre and postsynaptic membranes, leading to synaptic maturation.

The significant inverse correlation between FoxM1 expression and human epidermal growth factor 2 (HER2) (Bektas et al, 2008) suggests overexpression of FoxM1 may result from this interaction or that the p53mut R280K in MDA-MB-231 cells may not allow binding to the promoter region of FoxM1.

It may also allow binding to both genomic RNA and nascent mRNA transcripts, contributing to intragenic antitermination, by prevention of premature dissociation of the polymerase during transcription.

The surface representation suggests that like PTP1B, HD-PTP has an open binding pocket in its active site that may allow phosphate binding.

A K997A substitution in the Walker A box (WA) should prevent ATP binding, whereas a D1121A substitution in the Walker B box (WB) may allow ATP binding, but should be defective in ATP hydrolysis.

In the absence of Id1, these genes were significantly upregulated (Figure 6A), suggesting that the loss of Id1 may allow increased binding of Mitf to PU.1 and NFATc1, subsequently resulting in increased expression of target genes.