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When bone morphogenetic protein (BMP) is delivered to matrices in vivo may affect tissue engineered bone constructs for jaw reconstruction after cancer surgery.
Thus, a direct role of microbes in creating energy surpluses suggests that microbes may affect tissue trade-offs [8] and development.
Improvement of these hemodynamic and oxygen derived variables may affect tissue oxygenation and as a result preserve organ function.
Differences in lipid distribution and lipid class profile, as well as lipid dynamics like fat accumulation and fasting, may affect tissue concentrations of POPs [ 4, 13].
Additional differences include posttranslational modifications such as glycosylation, sialylation, and phosphorylation of mannose residues, which may affect tissue distribution and antibody production rates for the two enzymes.
Since fibroblasts play a structural role that is necessary for proper lung integrity, the presence of senescent fibroblasts may affect tissue microbalance and structural maintenance of the lungs.
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Physiological factors such as liver fluke (Fasciola hepatica) infection, breeding status, and in-tissue element interactions may also affect tissue concentrations, though their effects in red deer are not well understood.
It may even affect tissue that is beneath the final layer of your dog's skin.
As a tissue-engineering scaffold, it is speculated that the crosslinking degree of an acellular tissue may affect its tissue regeneration pattern.
Thus, understanding mitochondrial distribution, shape, and dynamics in all cell types is a prerequisite for developing and defining treatment regimens that may differentially affect tissues.
The theory is that fat tissue produces a weak form of estrogen that may affect breast tissue.
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