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Patients frequently travel long distances to obtain care for gestational trophoblastic neoplasia, which may affect cancer outcomes in these patients.
Artemisinin may affect cancer development and growth via various mechanisms.
CTC may affect cancer prognosis years before the onset of overt metastasis, improve the risk assessment, and help identify patients in need of treatment (Zieglschmid et al. 2005; Braun et al. 2005; Braun and Naume 2005).
Recent observations indicate that the timing of p53 re-expression during lung cancer development may affect cancer outcome [75], [76].
Additionally, the presence of HA may stimulate rises in extracellular Ca2+ concentrations, which may affect cancer cell behavior [43].
Given the known differential effect of EPHX1 alleles in the detoxification of procarcinogens, it has been proposed that these polymorphisms may affect cancer risk.
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Historically, these procedures were associated with significant complications, which may affect cancer-specific outcomes.
Bladder cancer is more common in older patients and this may affect cancer-specific survival (Walters et al, 2009).
As such, although targeting mitosis is currently being challenged as a flawed rationale to treat cancer (Komlodi-Pasztor et al, 2012), targeting of AURKA kinase activity may affect cancer-driving properties other than proliferation.
In population studies different individuals may be on different treatment regimens that may themselves affect cancer development and progression in different ways.
A recent Harvard study published online Oct. 17, 2012, in The Journal of the American Medical Association, found that multivitamins may also affect cancer risk.
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