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The theory is that fat tissue produces a weak form of estrogen that may affect breast tissue.
Other enzymes may affect breast cancer drug resistance, including those regulating drug inactivation or detoxification.
These findings suggest that systemic inflammation may affect breast epithelial neoplasia.
Altered glucose metabolism and elevated body weight may affect breast cancer (BC) prognosis [ 1].
The magnitude of effects seen in this study is meaningful and suggests that these variants may affect breast cancer risk.
The relevant time period during which 25(OH D levels may affect breast cancer recurrence or survival is currently unknown.
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Non-aspirin NSAIDs may also affect breast cancer survival, and NSAID use may correlate with ASA use.
Sodium sources alternative to NaCl improve feed utilisation, but they may adversely affect breast muscle traits.
Previous studies suggest that polycyclic aromatic hydrocarbons (PAHs) may adversely affect breast cancer risk.
If EDCs make breast cancer cells grow in the laboratory, for example, they may also affect breast cancer in women.
Hence, risk factors for BC may also affect breast biology and clinical behaviour, thus also BC prognosis.
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