Furthermore, positive mood induction may activate additional neural pathways associated with enhancing extinction learning (e.g., ventromedial/medial prefrontal cortex and anterior cingulate cortex; Phan, Wager, Taylor, & Liberzon, 2002).
The effectiveness of atropine, an anticholinergic agent, indicates that brevetoxins also activate neural pathways.
25, 26 These findings suggest that our cognitive training emphasizing virtual movement may activate the same neural systems involved in mobility.
An alternative possibility is that our screening may not have activated neural circuits related specifically to aggression.
Third, emotional stimuli may activate a network of neural structures, including the amygdala, prefrontal and sensory cortices, which in turn modulate attention and visuo-perceptual processing, resulting in enhancement of the subjective perceptual experience of threat cues and their ability to capture attention (e.g., Davis & Whalen, 2001; Vuilleumier, 2005).
Inhalation anesthetic sevoflurane may activate GABAA receptor to inhibit central nervous system, leading to apoptosis of neural degeneration, cognitive dysfunction in the critical period of brain development.
An arrest may activate the clause, or only a conviction.
Some neurons may activate a transcriptional cascade independent of FOS.
RONS may activate AMPK by direct an indirect mechanisms.
Or religious priming may activate concerns of supernatural punishment.
