TBARS is a marker of lipid peroxidation.
The maximum content of lipid peroxidation products (almost 1.4 times higher than in control) in these seedlings was observed on the fifth day after infection, which indicates the development of plant stress state caused by pathogenesis.
Moreover, 300 mM NaCl induced the maximum rate of lipid peroxidation in membranes and caused an increase in damage by 31% in leaves at 14 d when compared to the control.
The maximum intensity of lipid peroxidation was observed in variants with seed treatments with Ag nanoparticles and infection winter wheat seedlings of the Myronivska 808 variety and in seeds treated with Cu nanoparticles and an infection of winter wheat seedlings of the Renan variety.
Maximum anti-lipid peroxidation activity was observed by SCEE and lowest by SCAE with IC50 values of 119±1.0 μg/ml and 314±2.8 μg/ml respectively.
At the concentration of 1 mg/mL, the maximum inhibition level of lipid peroxidation is 57.61 ± 0.29% (ethanol extract), and for the ascorbic acid value it was 31.89 ± 1.24%.
Hydrogen peroxide induced lipid peroxidation in rat brain homogenates.
Another mechanism is lipid peroxidation.
Lipid peroxidation was quantified as malondialdehyde [21].
