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The pharmacodynamic parameters that were calculated for each clamp study included area under the curve of the glucose infusion rate (AUCGIR 0 300), maximum glucose infusion rate (GIRmax), and time to maximum glucose infusion rate (TGIRmax).
The PD parameters that were calculated for each clamp study included area under the curve of the glucose infusion rate (AUCGIR), maximum glucose infusion rate (GIRmax), and time to reach maximum glucose infusion rate (TGIRmax).
Using glucose infusion rate (GIR) versus time data, the maximum glucose infusion rate (GIRmax), time to maximum glucose infusion rate (tGIRmax), and total glucose infusion from injection to end of clamp (GItot) were calculated.
We did not find any correlation between the maximum glucose infusion rate required to maintain normoglycaemia and plasma insulin levels.
There was no correlation found between maximum glucose infusion rate to maintain normoglycaemia and plasma insulin level during hypoglycaemia (figure 1).
The pharmacodynamic parameters evaluated were area under the curve of the glucose infusion rate (AUCGIR), maximum glucose infusion rate (GIRmax), time to maximum glucose infusion rate (TmaxGIR), time to discontinuation of exogenous glucose infusion, time to half-maximal increase of peak action (Ti50), and time to half-maximal decrease from peak action (Td50).
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CI = confidence interval; CPC = Cerebral Performance Category; Emax = maximum measured glucose infusion rate; EC50 = insulin concentration associated with the half-maximum glucose infusion rate; ICU = intensive care unit; OR = odds ratio; SAPS = Simplified Acute Physiology Score; TK/TD = toxicokinetic/toxicodynamic.
During the course of poisoning, TK/TD relationships between the glucose infusion rate (E) and insulin concentrations (C) fit the Emax model E = (Emax × C / EC50 + C), where Emax is the maximum measured glucose infusion rate and EC50 is the concentration associated with the half-maximum glucose infusion rate.
Toxicokinetic/toxicodynamic relationships between glucose infusion rates and insulin concentrations fit the maximum measured glucose infusion rate (Emax) model (Emax 29.5 [17.5 to 41.1] g/hour, concentration associated with the half-maximum glucose infusion rate [EC50]46[35to to 161] mIU/l, and R2 range 0.70 to 0.98; n = 6).
Cmax, maximal observed concentration; EC50, concentration associated with half-maximal effect; Emax, maximum possible measured glucose infusion rate; R, correlation coefficient for the TK/TD model; T1/2, half-life; TK/TD, toxicokinetic/toxicodynamic.
Study 1: PK and PD were markedly different between BIAsp 30 OD and BHI 30 OD: the maximum insulin concentration and glucose infusion rate (GIR) were higher for BIAsp 30; time to maximum metabolism was 1.7 h sooner for BIAsp 30.
More suggestions(15)
maximum norepinephrine infusion
maximum glucose concentration
maximum air infusion
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maximum glucose formation
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maximum glucose recovery
maximum glucose level
maximum glucose uptake
maximum glucose conversion
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