As shown in Figure 2-A, maximums of cell proliferation were observed at a concentration of 1 mg/ml of aqueous extract (86.06%), ethyl acetate extract (126.22%), methanolic extract (249%) and TOF-enriched extract (260.11%).
Glutathione content tended to increase to a maximum during exponential cell proliferation, and then decreased at different rates as the cells approached plateau phase.
Maximum inhibition in cell proliferation was observed upon incubation of the cells with 0.5 μg ml−1 VP-16 and 20 μg ml−1 MXF for 72 h (up to 52±0.5%), compared to VP-16 alone (P<0.001).
In particular, oscillatory solutions may occur when δ, the maximum rate of occlusion, is sufficiently high; if δ exceeds the maximum rate of endothelial cell proliferation, η0, then the co-existence steady state may be unstable and periodic oscillations are possible.
We found that IL-10 was highly produced in co-cultures of MoDCs isolated ex vivo between days 3 and 17 pi and T cells of a naïve animal (average value of 720 pg/ml in FMDV infected pigs versus 250 pg/ml in naïve animals) (Fig. 5B), when the maximum inhibition of T cell proliferation was observed (see figure 4B).
These studies collectively support the idea that cell radius cannot exceed the maximum length of microtubules for cell proliferation.
The maximum effects of thrombin on cell proliferation and progression to S phase were noted at 10 units/ml.
A maximum of 80% inhibition of cell proliferation was observed at concentrations over 600 μg/mL for both MTT and LDH assays.
In contrast, another environmental pollutant, β-HCH, induced a marked concentration-dependent induction of cell proliferation, reaching maximum RPE of 61 ± 2 upon treatment with a concentration of 2.5 μM.
