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Cells incubated in culture medium only (without the compound) served as the control for cell viability both for the illuminated plates and for the ones kept in the dark; whereas dimethyl sulfoxide (DMSO; 50%, v/v) was used to observe maximum cell death (positive control).
Maximum cell death was induced by 250 μg/ml of FA as observed by Evans blue uptake.
In this study, maximum cell death was shown in pathogen inoculated leaves followed by Fol + SA treated leaves as compared with other treatments (Figs. 2c f, 3b).
However, it appeared that cell death induced by BE is more efficient after 12 hours when compared to BetA and maximum cell death is achieved after 24 hours (Figure 1C).
The highest concentration of UF008/SAP (1000 pg UF008/SAP /µl) caused maximum cell death whereas the same concentration of the non-immunized IgG/SAP conjugate did not cause any cell death (Figure 1).
Maximum cell death was achieved with all combined treatment groups at 48 h.
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Overall, PE extract induced the maximum cancer cell death followed by in ACE and MeOH extract.
We have previously demonstrated that the localization of hFasL within rafts is essential for maximum induction of cell death.
They achieved maximum oxidative burst and cell death levels producing normal HR lesions, but evidenced premature defense activation.
In an attempt to optimize treatment strategies we have focused on determining how anticancer agents can be combined in order to induce maximum levels of tumour cell death.
In particular, before and during the maximum ROS accumulation that precedes cell death, and at a late HR phase already manifesting cell death.
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maximum cell temperature
maximum cell loss
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maximum cell edge
maximum cell load
maximum chondrocyte death
maximum cell speed
maximum cell viability
maximum cell mass
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maximum cell survival
maximum cell area
maximum cell density
maximum cell size
maximum cell retention
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