Sentence examples for maximal stimulation and from inspiring English sources

Also, studies have shown that PPAR and RXR agonists, when used together to simultaneously activate the heterodimer complex, have synergistic effects allowing for maximal stimulation and expanding possible treatment paradigms (Papi et al., 2009; Yamanaka et al., 2012).

Muscle bundles were divided into two groups, one for the establishment of the force-length relation during sub-maximal and maximal stimulation, and the other for measurement of staircase and posttetanic potentiation.

The model provides a simple molecular explanation for the catalytic-like action of the polymerase, recapitulates the modest maximal stimulation, and rationalizes how a polymerase can be built from domains that bind preferentially to a microtubule-incompatible conformation of αβ-tubulin.

Drug-stimulated ATPase activity was analysed by the equation V=Vbas+ (Vmax×Sb)/(Sb+Ksb), where S is the concentration of stimulatory drug, V is the rate of ATP hydrolysis, Vbas is the ATPase activity in the absence of drug, Vmax is the maximum drug stimulated ATPase activity, Ks is the concentration of drug required for half-maximal stimulation, and b is the Hill coefficient.

Gmax and C50GLY denote respectively the maximal stimulation effect and the Ep concentration that produces 50% of the maximal response.

In contrast, CNP increased SO4 incorporation in a dose-dependent manner with maximal stimulation at 100 and 1000 n M (P < 0.001; Figure 2d).

Through titration experiments, a concentration of 10 n M AED was found to provide maximal stimulation of LNCaP, and no proliferation of LNCaP cells was observed without the addition of AED (data not shown).

To determine whether doxorubicin would induce the phosphorylation of JNK and p38 MAPK in macrophages deficient in ZAK, we employed a supramaximal dose of doxorubicin (500 μM) in order to determine the extent to which maximal stimulation of JNK and p38 MAPK would depend on the presence of ZAK.

The combination of estrogen and EGF resulted in maximal stimulation of cell migration and ERK phosphorylation.

In this study, the HD patients demonstrated a significant structural deficit in CD at each level of measurement including basal (non-stimulated), post-occlusion hyperemia and maximal stimulation with venous occlusion.

Purified TLR agonist controls, LPS and FSL-1, had optimal and maximal stimulation at 6 hours (Figure 1B).