Sentence examples for maximal proliferation rate from inspiring English sources

Controls were considered as cells with a maximal proliferation rate (100%).

However, addition of EGF, which induced a near maximal proliferation rate, resulted in a significant increase in DDX21 protein expression that was not further affected by insulin.

Even with such a high knockdown efficiency, we did not observe significant difference in the maximal proliferation rate between Si-C cells and Si-PKM cells.

The saturation of the cell density as well as the maximum proliferation rate decreased, although we found no significant changes on the half maximal time for proliferation.

Since we expect that the rate at which the tumour population increases by cell division is a saturating function of oxygen [ 21], we assume that the proliferation rate is of Michaelis-Menten type (see e.g. [ 20]), given by where β is the maximum proliferation rate per unit of cell density and s β is the level of oxygen at which the proliferation rate is half-maximal.

Other parameters were assumed to be cell type specific: I E, I R are the stochastic inputs of Teff and Treg cells respectively; α E, α R are maximal proliferation rates; γ E, γ R the clearance rates.

The constant k was calculated for each cell line between 24 and 72 h, the period of time in which the cell proliferation rate was maximal.

We have demonstrated that the over-expression of the c-myc gene in immortalised CHO cells can increase proliferation rate and maximal cell density in batch culture compared to the control.

And it was calculated using the following equation: −pEC50 = log Cmax − log 2 × (∑P − 0.75 + 0.25Pmax + 0.25Pmin), where Cmax = maximum concentration, ∑P = sum of proliferation rates, Pmax = maximum value of proliferation rate and Pmin = minimum value of proliferation rate [20 22].

The constitutive over-expression of c-myc, has clearly resulted in an overall increase in proliferation rate but also an increased maximal cell number under batch conditions.

Expression of SNTA1 and wild-type P66shc in HBL-100 cells resulted in a significant increase in H2O2, as well as an increase in the cell proliferation rate; combined expression of both these proteins resulted in maximal ROS generation and proliferation in these cell types.