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Both 60 and 80 kDa cytoplasmic bands increased from G1 to G2 with a detectable increase 2 hours after release, and a maximal increase after 8 h.
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Neither the Crs nor the Pplat reached during PV measurement was correlated with the maximal increase in Pao2 after PV measurement using both methods (data not shown).
The maximal increase in Pao2 after PV measurement using both methods was not different between patients with diffuse, lobar, or patchy ARDS (data not shown).
The maximal increase in Pao2 after PV measurement taken using both methods was compared between patients with diffuse, lobar, or patchy ARDS using one-way ANOVA.
In TNF- α-treated SCAPs, phospho-I κB α was obviously elevated in a time-dependent manner and phosphorylated P65 rapidly reached a maximal increase within 15 minutes after TNF- α stimulation and 2(b)).
Post hoc analysis revealed a maximal increase in skin blood flow after PACAP38 and VIP 5 min after injection, which was larger than compared to placebo (P < 0.001).
Using pom1 as a proof-of-principle, we observed a maximal increase of Purg1-pom1 transcripts after uracil addition within less than 30 minutes, and a similarly rapid decrease after uracil removal.
The AT1001 potency (EC50 value) and maximal increase in α-Gal A activity after AT1001 incubation were determined for each transiently expressed mutant form (Fig. 1).
We demonstrated that nor-NOHA decreased both the maximal increase in tissue resistance and elastance after ovalbumin challenge in sensitized animals, suggesting that arginase 2 contributes to the modulation of lung parenchyma specific hyperresponsiveness in this experimental model.
The maximal increase in CPP occurred in both groups after 30 min (mannitol +18%; hypertonic saline +27%; p < 0.05).
Maximal decrease of the CHL1 mRNA level was 20-fold, maximal increase was 34-fold.
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