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Only probe-sets and transcripts with a maximal expression level >5 (in log2 scale) were used.
In nTregs, the maximal expression level was reached after 24 h of activation and remained at this moderate plateau for the duration of the experiment.
The MAXC is the maximal expression level in the panel of M2 normalized cancer environments; MAXN is the maximal expression level among M1 normalized disease-free tissue environments, AV is the average level in the norm (average of M1 experiments) and VULNERABLES is the average level measured in the sub-panel of normal tissues most often suffering from side effects of therapy.
After submission, the resultant image illustrates the maximal expression level of the gene or genes of interest on a small representative graph of the distribution of average expression levels for each of ∼22814 genes in the given data set.
Interestingly, the lowest expression level of cytochromes containing heme a were repeatedly found in photoheterotrophically growing cells, whereas in cells grown in SYPG medium under microaerobic conditions a maximal expression level of cytochrome aa3 was observed and estimated to be 0.03 nmol/mg (dw).
β a is the maximal expression level of promoter.
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Nevertheless, the largest cluster of genes (cluster 1) identified differentially expressed genes upregulated with maximal expression levels at 12 to 16 weeks of age, or the time of detectable inflammation.
Consistent with this, the hepatoblast transcripts, α-fetoprotein (AFP) and transthyretin (TTR) became expressed between d6 and d10, reaching maximal expression levels at d14.
Our previous work [14] showed that at ten hours post infection both genes have reached approximately maximal expression levels.
While MX1 reached maximal expression levels after 9 hours, OAS1 was induced most strongly after 24 hours (Fig. 3A, B).
Thus maximal expression levels in the central domain of 0.56, 0.47, 0.68 and 0.2 for ArmS10, ΔNArm1 155, Myr-ΔNArm1 155 Myr-ΔNArm1 155mutands, respectively, only provided ArmΔCXM19stimutants
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