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Additionally, we predicted that senescence and maturation would influence the temperature-induced CSR by affecting metabolism and protein biosynthesis when compared with fish that were less mature.
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The betatron would influence all subsequent accelerators.
Thus, helping formalize a molecular signature for cardiac myocyte maturation would advance the field.
Identifying how these combined factors are impacted by maturation would benefit current risk screening and prevention efforts.
These results indicated that longer periods of maturation would induce a higher degree of migration.
In fact, as shown here, affinity maturation would tend to impair the antibody potency.
In any case the maturation would have affected the intervention and non-intervention groups equally.
Because a reduced ability to arrest phagosomal maturation would probably affect intracellular survival, we conclude that phagosomal maturation arrest of the mptC mutant is not significantly affected.
A single-trait mixture model for maturation would not be able to distinguish between the effects of health and sexual maturation (i.e., the binary sexual maturation status being affected by the binary and unobserved health status).
Thus, in skeletal tissue affected by these cellular metabolic disturbances, altered growth expression rates would expectedly influence bone maturation (growth) indices as demonstrated by the db/db and ob/ob mutant groups.
Monocytes, however, were more sensitive to VIP than macrophages, and as such would also suggest that maturation might influence VIP responsiveness through modulation of receptor expression.
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