Sentence examples for maturation of pre from inspiring English sources

ER is one of the major membrane sources contributing to the maturation of pre- or autophagosomal structures and thus structurally connected to the autophagic machinery (Hayashi-Nishino et al., 2009; Hailey et al., 2010).

The ER, mitochondria as well as plasma membrane are the major membrane sources contributing to the maturation of pre- or autophagosomal structures (Hayashi-Nishino et al., 2009; Hailey et al., 2010; Ravikumar et al., 2010).

Moreover, the failure to segregate PrE and epiblast cells suggests the maturation of PrE cells could be linked to their organisation as a monolayer at the ICM surface.

These results indicate that, although aPKC inhibition does not cause a switch in cell fate from PrE to epiblast, it compromises the maturation of PrE cells.

We show that inhibition of aPKC from the mid blastocyst stage not only prevents sorting of PrE precursors into a polarised monolayer but concomitantly affects the maturation of PrE precursors.

We propose a mechanism whereby aPKC translates positional information to resolve the 'salt and pepper' pattern by promoting both maturation of PrE and sorting of PrE and epiblast cells into separate layers.

Waberski et al. [ 10] suggested that cytokines in the uterine lymph, when induced by semen, may undergo counter-current transfer to the ovary and thereby accelerate the final maturation of pre-ovulatory, mature follicles.

The maturation of pre-60S is coupled to the dynamic association of approximately 90 assembly factors, hierarchical incorporation of RPLs and sequential processing of ITS1 and ITS2.

They are not strictly required for rpS5 function in nuclear export of SSU precursors, but are crucial for final cytoplasmic 3' maturation of pre-18S rRNAs.

Interestingly, McLaughlin et al. [42] and Pasarica et al. [43] reported increased small adipocytes in subcutaneous fat from obese and type-2 diabetic subjects in two independent studies, suggesting the presence of an impairment in adipose cell differentiation and early maturation of pre-adipocytes linked to obesity-associated co-morbidities (such as insulin-resistance and type-2 diabetes).

To delineate further the role of Chd1 in transcription, we examined the maturation of pre-mRNA to spliced RNA since Chd1 associates with elongation factors as well as splicing components, and consequently, depletion of Chd1 impairs the splicing efficiency [41], [42].