Sentence examples for maturation of erythroid from inspiring English sources

Sandoval, H. et al. Essential role for Nix in autophagic maturation of erythroid cells.

Sandoval H, Thiagarajan P, Dasgupta SK, Schumacher A, Prchal JT, Chen M, et al. Essential role for Nix in autophagic maturation of erythroid cells.

Thus, miR-451, which belongs to this group, was shown to regulate maturation of erythroid cells [38].

For example, NIX has been shown to play an essential role in the selective mitochondrial macroautophagy observed during the maturation of erythroid cells [37], [38].

These observations suggested that some erythroid precursors had either died after exposure to hemozoin on day 0 of culture or that hemozoin may have delayed the maturation of erythroid precursors.

In contrast, AML1-ETO and PML-RARA did not show a significant effect on the numbers and maturation of erythroid cells in spite of their effects on erythroid colony numbers.

It is primarily involved in enhancing the survival rate and maturation process of erythroid cells and acts at least in part by upregulating Bcl2l1.

In order to exclude that the maturation defects of erythroid cells in apparently unaffected nb/nbl morphants could be due to a generalized developmental delay we performed Wright-Giemsa staining on blood smears at 3 dpf.

Stat5a and Stat5b were expressed during both primitive and definitive erythropoiesis, but exhibited increasing expression during the maturation of primitive erythroid cells and the opposite pattern during the maturation of adult definitive erythroid cells.

Although Src kinases are activated by JAK2 in erythroid cells upon Epo stimulation [28] and a Src inhibitor impaired Epo-independent maturation of PV erythroid progenitors [13], we demonstrated that polycythemia induced by JAK2 V617F was independent of Lyn, Hck, and Fgr, the three principal Src family kinases expressed in myeloerythroid cells.

For the functional relevance it is essential that two similar cell lines, K562 and KG1a, representing blockage at an early stage of maturation of the erythroid and myeloid lineages [21] showed a similar metabolic response to the MPA+BEZ treatment whereas HL-60 cells, which are closer to the mature granulocyte stage [21] showed a different overall response.