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The age at maturation is thought to be 2 3 years, and the maximum lifespan is at least 8 years.
Although timing of maturation is thought to evolve as a consequence of selective pressures [ 63], which implies restrictive environmental forces acting on organisms, here we show that the environment, rather than restricting the ontogeny of organisms, can act inducing changes in life-history traits, which may also lead to population and evolutionary changes.
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White matter injury and abnormal maturation are thought to be major contributors to the neurodevelopmental disabilities observed in children and adolescents who were born preterm.
As developmental factors (e.g., cognitive maturation) are thought to have an impact on anxiety in youths [ 47], it may well be that the predictors of change produced by CBT vary as a function of children's age.
In hydrogenase maturation, HypB is thought to carry out both the insertase and metallochaperone roles in most organisms due to the presence of a histidine stretch located at the N-terminus of these proteins.
In oysters, synchronized gonad maturation and spawning is thought to occur via environmental cues, including tides and pheromonal cues released by conspecifics that initiate endogenous cues [ 33].
The function of maturation stage ameloblasts is thought to be primarily one of the ion transport/regulations to facilitate secondary crystal growth, completing enamel mineralization to produce a hard, highly mineralized tissue (35).
While the maturation of myelin is thought to follow a similar spatial-temporal pattern of cognitive and behavioral development [Casey et al., 2005; Fields, 2008], the association of these structural and functional aspects remains poorly understood.
The Atg14L complex functions in autophagosome formation, the UVRAG complex functions in autophagosome and endosome maturation, while the Rubicon complex is thought to suppress autophagosome and endosome maturation.
On the contrary, IFN-α produced by lymphoid dendritic cells is another important maturation factor for dendritic cells and is thought to be involved in SLE pathogenesis [ 48].
This increase in white matter volume is thought to reflect ongoing maturation of neuronal axons, for example, myelination and/or increasing axonal caliber.
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