However, some studies have also implied that organic matter amendment may induce unwanted changes of tailing pore water chemistry, such as increased metal release by municipal biosolids [14] and cattle manure [15, 16].
Specifically, exposure to particulate matter air pollution may induce or exacerbate anxiety through increased oxidative stress and systemic inflammation 9 10 11 12 13 14 15 16 17 or through promotion or aggravation of chronic disease.
The inflammatory response to particulate matter in welding fumes may induce oxidative stress in the lung (Valavanidis et al. 2013).
However, the A3R antagonists revealed the presence of A3R in the white matter, and this may indicate the potential for imaging neurological diseases involving the white matter, as suggested by González-Fernández et al. who found that activation of A3Rs may induce white matter death [ 46].
These regions have historically had higher levels of air pollution, and our evolving understanding of RA pathogenesis suggests that inhaled particulate matter, similar to cigarette smoke, may induce local lung inflammation as well as systemic inflammation.
Particulate matter, Fe, Cr, and other metals in inhaled welding fumes may induce the formation of ROS and thus oxidize nucleobases such as guanine of RNA and DNA or from the intracellular nucleotide pool.
Midazolam infusions may induce tolerance and a withdrawal syndrome in a matter of days.
Shear deformation mechanochemically may induce a change in the molecular structure of organic matter (e.g., Fulton and Harris 2012).
Background: Laboratory studies suggest that fine particulate matter (≤ 2.5 µm in diameter; PM2.5) can activate pathophysiological responses that may induce insulin resistance and type 2 diabetes.
It appears that elevated CO2 often increases the total amount of carbon supplied to below-ground microorganisms and may induce "priming" effects to accelerate the decomposition of old or recalcitrant organic matter.
