Sentence examples for matter changes described from inspiring English sources

In view of the cognitive, olfactory, auditory, vestibular, and volumetric white matter changes described in exposed children, a series of critical questions arise: What is the role of PM in the neuroinflammatory process described in highly exposed children?

Yet, the magnitude of 'pain-related neglect-like symptoms' is less compared with the neglect of stroke patients, possibly because grey matter changes described in pain are considerably smaller than they are in stroke.

In the same stream of thought, the central delay in the brainstem auditory evoked potentials and the significant white matter volumetric changes described after 1-year followup of MCMA versus control children could be related to the accumulation of abnormal proteins in key neuronal groups and the significant neuroinflammation involving both gray and white matter [ 30, 35, 67].

Accordingly, our patients with Tourette syndrome with OCD and concomitant ADHD show orbitofrontal cortex grey matter reductions, thus reversing the pattern of cortical volume change described in 'pure' OCD.

Subclinical, possible ischemic, white matter lesions [39 43] and grey matter changes [44, 45] have also been described in migraine patients.

Previous reports have not described any specific changes on the imaging studies whereas this report shows periatrial and periventricular white matter changes consistent with delayed myelination.

A growing body of literature is emerging that describes the use of diffusion-weighted MRI to detect microscopic grey matter changes in a number of neurodegenerative disorders.

As described above, elevated glutamatergic transmission within discrete anatomical circuits also may explain the targeted nature of gray matter changes within mood disorders (e.g., affecting left more than right sgACC) (McEwen and Magarinos 2001; Drevets and Price 2005).

Frontal-lobe accentuated white matter changes in myotonic dystrophy type 2, which correlated with age, may support this hypothesis since fractional anisotropy reduction in frontal regions had been described in normal ageing previously (Damoiseaux et al., 2009).

In the following paragraphs we describe PrP deposits in cortical grey matter, followed by changes in the subcortical white matter, cerebellar grey and white matter changes and the co-localisation of abnormal PrP with myelin and axonal structures.

Typical white matter changes on MRI (T2W)  C.