In order to compare the difference between the control matrix and condition-specific matrices we quantified the drift using the Frobenius matrix norm of the difference between the matrices.
To investigate whether inhibition of cell death induced by iPTH is associated with bone matrix modifications, we quantified the prevalence of osteocytes death in cortical bone stained using the TUNEL assay.
Considering that β-1 integrin is the collagen type I binding receptor on cell membranes and mediates cell matrix adhesion, we quantified the β-1 integrin expression level in cells on various matrices at 12 h post-plating.
Using thin films of extracellular matrix proteins, we have quantified, on a cell-by-cell basis, phenotypic parameters of cells on different extracellular matrices.
To quantify differences between matrices, we compute the basic reproductive number (R_{0}) defined as the dominant eigenvalue of the next generation matrix [5], which is equivalent to the dominant eigenvalue of the contact matrix up to a constant.
We found that to calculate the stresses accurately in these settings, we had to account for mechanical heterogeneities within the matrix, which we visualized and quantified using confocal reflectance and atomic force microscopy.
Finally, MSCs were induced to undergo osteoblastic differentiation on the three materials, and deposition of a mineralized matrix was quantified.
The cross-sectional nonlinearity accounts for matrix cracks, quantified in terms of crack density and delamination, quantified in terms of the delamination width.
In this research, the morphological matrix is quantified by associating the properties of function solution principles with the information of customer preferences and product failures.
The mechanical capacity reduction is attributed to the incorporation of the soft/compliant silver phase and the subsequent graphitization of the amorphous carbon matrix, as quantified through XPS.
