Sentence examples for matrix interaction in from inspiring English sources

We employed similar methodologies to elucidate the molecular mechanisms of integrin and extracellular matrix interaction in regulating leukocyte function.

It could also be seen that there is lower swelling number associated with the NR composite filled with 10-μm MS as compared to those filled with 20- and 75-μm MS. This is due to better dispersion of smaller microsize of MS particle in NR, promoting better filler-rubber matrix interaction in NR composites [47, 48, 49].

CD44s and CD44v have been shown to mediate the cell-extracellular matrix interaction in various biological processes such as lymphocyte homing [ 12], hematopoiesis [ 13], embryogenesis [ 14], and wound-healing [ 15].

Since the migratory and metastatic potential of cancer cells is fore timed by their interaction with endothelial cells and ECM proteins like collagen [ 16], we assessed the effect of Yukyung Karne treatment on cell to matrix interaction in vitro by collagen binding ability using ovarian cancer cells.

Although the importance of extracellular matrix interactions in metastasis is well documented, systematic approaches to identify their roles in distinct stages of tumorigenesis have not been described.

Given the BST-2-independent effect of B49Mod1 in blocking immune cell to cancer adhesion, we developed a system to evaluate the effect of B49Mod1 on other adhesion molecules that may mediate cell: matrix interactions in the absence of BST-2.

The biofunctional network we have designed based on this knowledge may be useful as a defined niche for regulating ESC pluripotency through selective cell matrix interactions, and the method we present may be more generally useful for probing matrix interactions in stem cell self-renewal and differentiation.

Adhesive molecules play important roles in ligand binding, cell-cell and cell-extracellular matrix interactions in higher eukaryotes [28], [29].

While traditional traction force measurements have provided quantitative information about cell matrix interactions in two dimensions, recent studies have shown significant differences in the behavior and morphology of cells when placed in three-dimensional environments.

Similarly, the over-representation of genes involved in cell adhesion (Fig. 4A), including several with actin-binding and structural molecule activity (Fig. 4B), suggests a particularly important role for the regulation of cellular, neuritic, and synaptic morphology through cell-cell and cell-extracellular matrix interactions in HVC.

As increased ligand density might be another mechanism behind altered cell-Fn matrix interactions in tumors, future studies are needed to probe how antagonizing RGD binding sites could alleviate (in a dose-dependent manner) subsequent cell behavior in tumors.