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Several other methods for processing the data are proposed such as compressing the matrix, creating a distance matrix, calculating distances, etc.
As a general rule, NCs are prepared by dispersing a nanometer-sized filler into a polymer matrix creating a network able to improve the properties of a host polymer.
Through an iterative procedure, this method accomplishes a rescaling of the original edge weights matrix, creating a bistochastic matrix in which all columns and rows sum to one [17].
Physiologically, bone remodeling starts with osteoclastic bone resorption characterized by dissolution of the inorganic matrix of the bone followed by enzymatic degradation of the organic component of the matrix, creating a complex three-dimensionally structured surface.
These complexes increase the viscoelastic nature of the pericellular matrix, creating a highly malleable extracellular environment which supports a cell shape change necessary for cancer cell proliferation and migration [ 30- 32].
Studies by Lin et al. [ 6] showed a 110% increase in interfacial strength for ZnO nanowire coated carbon fibre which was suggested to be due to the ZnO nanowires penetrating the epoxy matrix creating a strong interlock, and further modification of the nanowire dimensions resulted in the interfacial strength increasing by up to 228% [ 37].
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During substrate oxidation, protons are pumped out of the mitochondrial matrix, creating an electrochemical proton gradient.
Encapsulating cells within a polymer matrix creates a three-dimensional (3D) scaffold that may more accurately represent the native microenvironment and cell organization.
Hence, crosslinking ECM protein with synthetic matrix creates a stable scaffold with tunable mechanical properties and with long-term cell anchorage points, thus supporting cell attachment and growth in the 3D environment.
We show that the physical incorporation of Tβ4 in this bioactive matrix creates a three-dimensional (3D) environment conducive for human umbilical vein endothelial cell (HUVEC) adhesion, survival, migration and organization.
A novel mechanism of biofilm-associated antibiotic resistance has been described recently in P. aeruginosa: released DNA, the highly anionic polymer working as a cation chelator in the extracellular matrix, creates a localized cation-limited environment.
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