As mechanical properties are closely related with the dispersion of the filler within the matrix, we quantified the dispersibility by analyzing frequency distributions of light intensity transmitted through film samples.
To investigate whether inhibition of cell death induced by iPTH is associated with bone matrix modifications, we quantified the prevalence of osteocytes death in cortical bone stained using the TUNEL assay.
To quantify differences between matrices, we compute the basic reproductive number (R_{0}) defined as the dominant eigenvalue of the next generation matrix [5], which is equivalent to the dominant eigenvalue of the contact matrix up to a constant.
Considering that β-1 integrin is the collagen type I binding receptor on cell membranes and mediates cell matrix adhesion, we quantified the β-1 integrin expression level in cells on various matrices at 12 h post-plating.
Using thin films of extracellular matrix proteins, we have quantified, on a cell-by-cell basis, phenotypic parameters of cells on different extracellular matrices.
Infiltrated endothelial cells and macrophages in Matrigel matrices were quantified in a single-cell suspension by fluorescence-activated cell sorting (FACS) (Figure 1B, Figure S1A).
The elastin content of native tissue and acellular matrices was quantified using the FASTIN elastin assay (Biocolor, UK) according to the manufacturer's instructions.
The sulfated GAG (GAG) content of native tissue and acellular matrices was quantified using the Blyscan GAG Assay Kit (Biocolor, UK).
The differences between these two coherence matrices were quantified by computing the maximum cross-correlation for each set of coherence matrices for the figure and the ground stimuli respectively.
Additionally, the Mantel test can be used for comparing any similar rank matrices, as we illustrate by quantifying the relationship between topological overlap and spatial co-localization, both of which can be represented as matrices of pairwise interactions.
