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Other tissue-engineered grafts of decellularized matrices have recently been tested in clinical trial.
Such matrices have recently become available [25].
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The maximum entropy completion problem for block-circulant matrices has recently been solved by the authors, although leaving open the problem of an efficient computation of the solution.
A number of short sequences in proteins located in the extracelluar matrix have recently been identified to play important roles for bone regeneration, including osteoblast proliferation, migration and differentiation[6] [9].
A convex relaxation of the rank minimization problem by minimizing the nuclear norm instead of the rank of the matrix has recently been proposed.
Ca2+ influx to the mitochondrial matrix has recently been implicated as a mechanism to regulate respiratory bioenergetics by allowing the electrochemical potential across the membrane to transiently "pulse" from ΔΨ-dominated to pH-dominated, facilitating a reduction of oxidative stress.
A laminin-rich matrix has recently been shown to promote differentiation of mouse fetal hepatoblasts both in vitro and in vivo [71], supporting our findings of higher levels of albumin production when HFH are cast in gels with a mLBM.
Metal organic frameworks (MOFs) containing mixed matrix membranes have recently received increasing attention because of the favorable compatibility with polymer chains.
In addition to their role in facilitating tumor invasion and angiogenesis, matrix metalloproteinases have recently been assigned a role in helping block tumor progression and angiogenesis.
While the structure and function of a number of shell matrix proteins have recently been characterized [ 7, 10- 17], the regulatory mechanisms that govern these shell-building processes remain largely unknown.
Because the mast cell contains potent mediators, including histamine, its potential contributions to the processes of inflammation and matrix degradation have recently become evident; thereafter, we investigated the potential protective effect of histamine deficiency against aggrecan-induced arthritis.
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