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Tumor formation is multifocal, stochastic and matches the transgene expression.
NIR laser irradiation in the presence of ligand triggered 3-dimensional patterns of transgene expression faithfully matching the illuminated areas of plasmonic cell constructs.
Pathology propagated from transgene-expressing neurons to adjacent brain regions lacking any detectable transgene expression.
Plastid transformation offers the unique advantages of high-level transgene expression and increased transgene containment compared with conventional transgenic technologies.
Thus, administration of AII significantly increased tumor transgene expression.
In other words, the transgenic rice plants overexpressing 35S::OsNCED3 had transgene integration (based on antibiotic selection), but without transgene expression.
Adenoviral transgene expression was maintained throughout the differentiation process.
Higher transgene expression was maintained for at least 4 months.
To localize transgene expression, enhanced green fluorescence protein was used.
In transfected cells, such deletions reduced the transgene expression.
In the following, "phenotype" refers to compositional, nutritional, agronomic traits and level of transgene expression.
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