Sentence examples for marrow of aged from inspiring English sources

As previously indicated (Sherwood et al., 1998, 2000; Alter-Wolf et al., 2009), and shown in Fig. 1B, the remaining pro-B cells in the bone marrow of aged BALB/c mice are low in λ5 SLC and this is also observed for aged B6 mice (data not shown).

Moreover, we assessed the extent of co-localization between γH2AX-foci and telomeres in CD34+ and CD34− cells obtained from cord blood and bone marrow of aged donors.

Consistent with an immature phenotype, total NK cells and NK cell subsets from the bone marrow of aged mice expressed higher frequency of CXCR3.

We have previously reported a decreased number of total NK cells in the blood and spleen and reduced frequencies of CD27− CD11b+ mature NK cells in the blood, spleen, lymph nodes, and bone marrow of aged mice.

We wondered whether there is a similar increase of CD137+ CD4+ T cells in the bone marrow of aged CD137−/− and CD137L−/− mice, and whether the missing increase in myelopoiesis in these mice is due to a deficiency in the CD137+ CD4+ T cells or in the hematopoietic progenitor cells.

Because hematopoietic cells from young mice failed to mature properly in the bone marrow of aged mice, these data indicate a deficiency in the stromal cells of aged mice.

The present study was designed to assess the numbers/production of NK cells in the spleen and bone marrow of aging, normal mice, after in vivo dietary administration of E. purpurea (14 days), or, after injection of thyroxin, a stimulant of NK cell function (10 days).

Even with this major difference in design, our results with primary bone marrow adipocytes directly isolated from the bone marrow of aging mice agree remarkably well with their results.

NK cells expressing the inhibitory receptor KLRG1, which is normally expressed in the most mature NK cells (Huntington et al., 2007), were significantly reduced on total NK cells in the bone marrow and spleen of aged mice.

This finding seems to be unrelated to the increase in CD49b− NK cells in the bone marrow and spleen of aged mice because the latter are TRAIL−, and it has recently been shown that TRAIL+ liver trNK cells and conventional TRAIL− NK cells have different precursors (Daussy et al., 2014; Sojka et al., 2014).

There are very few published data on sex chromosome aneuploidy in the marrow cells of females with or without a haematological malignancy, but it appears that X chromosome aneuploidy is not a feature of the marrow cells of ageing women (Secker Walker 1971).