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Methylated H3K9 marks are recognized by epigenetic regulators in the heterochromatin proteins 1 (HP1) family, which is conserved in a large number of species [22].
Both marks are recognized by the chromodomain protein Pdd1p [ 58].
(A ) At loci of compact and repressed chromatin, H3K27-me3 marks are recognized by EED.
Existing H3K27me3 marks are recognized by EED and, possibly in cooperation with Ezh2's SANT domains, Ezh2 methyltransferase activity is increased.
H3K4me3 marks are recognized by chromatin remodeling factors facilitating transcription by altering the structure, composition, and positioning of nucleosomes, by components of the spliceosome, and by proteins involved in mRNA capping and stability [ 46].
Nevertheless, it is a little bit surprising that both AKT1-mediated up- and down-regulated genes have at least one differential binding site for H3K27me3 or H3K9me2 marks but there is no significant difference in enrichments since both histone marks are recognized as repressive marks.
Similar(54)
This methylation mark is recognized by the Tudor domain protein p100-TSN, which further suppresses apoptosis.
The phosphoacetylation mark was recognized by the 14-3-3ζ 14-3-3ζ 14-3-3ζ of proteinsereaderarks, and was thus profected from removal by phosphoserinease [ 77].
Tokens that are not recognized as chemical entities are marked with O. Tokens that are recognized as the beginning of a chemical entity are marked with B. Tokens that are recognized as continuing the name of a chemical entity are marked with I.
Short inverted repeat (IR) DNA sequences marking each end of the transposon are recognized by a DNA transposase (encoded by the transposon itself).
All of the following are recognized symptoms of clinical depression EXCEPT: a. Marked loss of interests.
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