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Exact(5)

The genome-wide distribution of H3K4me2 and H3K9me3 showed that these two marks are mutually exclusive (Fig.  5).

When two marks are mutually exclusive, such as H3K27ac and H3K27me3, this algorithm can maximize the appearance of such relationships.

Although the H4K16ac and H4K20me marks are mutually exclusive, the H4K12ac mark was recently detected along with the novel H4K16me1 mark.

Since they act on the same lysine residue, these marks are mutually exclusive, and the switch between methylation and acetylation has been well established.

A similar study by Brinkman et al. [ 4] found that H3K27me3 and DNA methylation are compatible except at CpG-rich promoters where the two marks are mutually exclusive, supporting a previous study by Komashko et al. [ 5].

Similar(54)

At promoters, high levels of H3K4me1 and active marks were mutually exclusive (Additional file 1: Figure S5A), and H3K4me1 was unable to recruit p65 on its own.

Moreover, virulence (var) genes are mostly poised and marked by a unique set of activation (H4ac) and repression (H3K9me3) marks, which are mutually exclusive to other Plasmodium housekeeping genes.

The two markets are mutually hostile.

All together, these results in patients' samples are in agreement with our previous observations in the DU145 cell line and support in vitro data showing that these two silencing marks are not mutually exclusive at CpG islands.

In Fig. 15 asterisks mark some splits that are mutually incompatible and include at least one of the long branch sequences.

Indeed, the H3K4 methyltransferases ASH1L [ 118] and SET7 [ 119] are less efficient in depositing H3K4me on histones marked with H3K9me in human cell lines, and H3K9me3 and H3K4me3 are mutually exclusive marks in mESCs [ 18].

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