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The productions of both markers were stimulated by the common latent variable.
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Figure 6: Flow cytometric analysis of surface activation markers on human monocytes stimulated with masked and non-masked LPS. 1 × 105 monocytes were stimulated as indicated.
(D) Lineage depleted PBMCs were stimulated with CpG A or R848 then analyzed by FACS for surface markers and IFNα intracellularly.
OSCs were stimulated with poly (I C) to induce IFNβ production and stained for the microglia marker Iba1, the astrocyte marker glial fibrillary acidic protein (GFAP) and the neuronal marker neurofilament-medium (NF-M).
(B) Lineage depleted PBMC were stimulated with LPS for 6 h, then the stimulated cells were collected and stained with surface markers followed by intracellular staining of TNFα.
Cells were stimulated 48 hrs post transfection.
C-reactive protein (CRP), an easily measured marker of systemic inflammation, is stimulated by other cytokines, especially interleukin-6 (IL-6).
Furthermore, the pool of undifferentiated cells was stimulated to express mesodermal markers associated with cardiac lineage differentiation.
This observation suggests that the resident, non-immune cells of the cochlea could be stimulated to produce inflammatory markers.
Alkaline phosphatase activity, a marker of cartilage maturation, was found to be stimulated by CT, SCT, HCT, PCT, and synthetic PCT increased alkaline phosphatase activity over 2-fold above that in cartilage incubated in medium alone.
The postmeiotic markers (Odf2, Act, and Prm1) [ 6, 15] are stimulated markedly.
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