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Nevertheless, it should be noted that the increases in the plasma levels of these markers were small.
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Most (58 %) of the intervals between markers were smaller than 10 cM (Fig. 3).
Most of the spaces between two markers were smaller than 20 cM on the genetic linkage map.
In Quartiles 2 and 3, improvements in CV risk markers were smaller compared with those in Quartile 1.
The mean interval between adjacent markers is 5.79 cM, and 85% of the intervals between adjacent SSR markers were smaller than 10 cM.
Further, pre- versus postmenopausal differences in bone turnover markers were smaller in the Chinese American versus white women, supporting the known racial differences in microarchitecture and fracture risk.
The regression slope obtained from mtDNA (with a negative value) and X-chromosome markers were smaller than those obtained from Y-chromosome SNPs.
Longest gaps between markers were smaller in Prunus, with only four exceeding 20 cM: 21.7 on PG7; 21.9 on PG6, 20.8 on PG8 and 28.6 on PG5.
Indeed, if the number of markers is small, the theoretical relationship matrix is used and the complete pedigree is needed for calculating this in a correct way.
The number of validated genetic treatment selection markers is small in number and applies to a fraction of health care.
Within these regions of minor rearrangements the distance between the markers was small both on the linkage map and on the BAC assembly.
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