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Exact(6)

These markers were originally developed for use in humans [ 63], but have been shown to be highly informative for use in chimpanzee population genetics studies [ 39, 64].

The DArT markers were originally obtained by cloning random fragments of genomic representations [ 16], a process that introduces some degree of marker redundancy.

All the 6,580 selected markers were validated primarily by gel- and sequencing-based assays using the genomic DNA of four chickpea accessions (ICC 4958, ICC 4951, ICC 12968 and ICC 17160), from which the InDel markers were originally detected.

These markers were originally developed for gene tagging in Brassica oleracea L. to specifically amplify coding regions of the genome with ambiguous primers targeting GC-rich exons (forward primers) and AT-rich promoters, introns, and spacers (reverse primers).

This included four accessions (ICC 4958, ICC 4951, ICC 12968 and ICC 17160) from which the InDel markers were originally identified, and 20 additional desi (9) and kabuli (11) accessions (Supplementary Table S1).

However, these markers were originally identified from changes in protein expression and it is possible that their differential expression is regulated at the protein level and therefore differs to their transcriptional profiles.

Similar(54)

The addition of SRAP markers to the framework map allowed the SSR markers that were originally unlinked or floating to be placed as accessory markers.

Unlike our assay of mtDNA variation, where both chimpanzees and humans were sequenced at comparable portions of the first hypervariable region of the mtDNA, we assessed chimpanzee NRY variation by typing microsatellite markers that were originally discovered as polymorphic in humans.

Clinical markers that were originally deranged can be followed, looking for improvement.

Of the 1010 markers that were originally scored in KxO, 93 loci had allele frequencies greater than 65%, and these are summarized in Additional file 13 for possible further analysis.

Note that the 26 markers analyzed here were originally chosen for other oncogenic studies conducted in our laboratory.

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