Although CHP itself did not show significant immune-enhancing activity, the inflammatory markers were enhanced when the adjuvants were included (TNF/CHP nanoparticles, free TNF, and CTB).
In addition, the combined delivery of TGF-β2 and GDF5 enhanced the expression of cartilage markers and collagen II protein content on substrates with isotropic porosity, whereas enthesis markers were enhanced in areas of mixed anisotropic/isotropic porosity.
The serum levels of ALT and BUN, which are specific toxicological markers, are enhanced in the case of liver injuries and kidney injuries, respectively [ 35, 36].
Moreover, as compared with animals that lack rac1 for 3 weeks only, the expression of fibrotic markers was enhanced in mice lacking rac1 for 15 months.
The specificity of this technique, besides the use of tumor-associated markers, is enhanced by visual appreciation of the digitalized cells.
Also, type I collagen and FSP-1 (an EMT marker) were enhanced in animals subjected only to UUO, which was not seen in the Hemin-treated group (Figure 3B, 3D, and 3E).
Levels of γH2AX, a DSB marker, were enhanced in both Parg-deficient ES cells and PARG knockdown MIAPaCa2 after MMS treatment.
Additionally, we found that surface expression of integrin αIIbβ3, an important megakaryocyte marker, was enhanced in CIB1 overexpressing cells as determined by flow cytometry.
The sensitivity of this simple marker was enhanced by the results of EMG assessment; conversely, the association of a sciatic nerve conduction block with the lack of foot flexion seemed highly specific of a prolonged duration of MV.
As indicated in our study, immunofluorescence analysis of breast cancer patient tissue also showed that markers for autophagy were enhanced in ER α-positive samples.
Compared with adherent-growing cells, markers for pluripotency, stemness and cancer progression, including the CSC surface marker c-Met, were enhanced in spheroidal cells.
